Alzheimer’s disease (AD) has been regarded as one of the most common neurodegenerative diseases, and is mainly manifested as progressive cognitive dysfunction, memory and learning decline, abnormal daily behavior, personality changes and decline in activities of daily living though its cause remains unclear. The main pathological features of AD have been revealed as deposition of amyloid β-protein（Aβ）, neurofibrillary tangles (NFTs), synaptic and neuronal loss etc., and these changes contribute to neurological inflammatory responses. The etiology and pathogenesis of AD are quite complex and diverse, and current most prevailing hypotheses include Aβ cascade hypothesis, abnormal phosphorylation of tau protein, nerve inflammation and so on. Much evidence showed that the inflammation response played important roles in the development and progression of AD. In this paper, we reviewed the relationship of some inflammatory cytokines［IL-1β, IL-6, IL-4, IL-10, transforming growth factor-β1 (TGF-β1), tumor necrosis factor-α (TNF-α) and others］with AD.